Cancer cell DNA doubling — when cells accidentally duplicate all chromosomes without dividing — now has a new risk factor: ...

A hidden clue may explain why some mutated cells become cancerous and others don’t: how fast they divide. A new study from researchers at Sinai Health in Toronto reveals that the total time it takes ...

Triple negative breast cancer (TNBC) is one of the most aggressive and hardest forms of breast cancer to treat, but a new study led by Weill Cornell Medicine suggests a surprising way to stop it from ...

A cell copies all six billion letters of its DNA, gears up to split, and then simply… doesn’t. It sits there, swollen with ...

Left: Normal cell division with the chromosomes (blue) lined up and ready to be pulled into two separate daughter cells by the two centrosomes (green). Right: In faulty cell division, too many ...

Scientists have uncovered a previously overlooked mechanism that may help melanoma cells become effectively “immortal.” ...

A new study by the Garvan Institute of Medical Research has uncovered a hidden mechanism explaining why breast cancer can return many years after successful treatment. Published in Nature ...

Scientists have uncovered a surprising twist in how cells behave when division goes wrong. Sometimes a cell successfully copies its DNA but fails to split into two, leaving it with double the genetic ...

Megan Sweet slices tumors. A normal day in the lab finds the Virginia Tech graduate student with hands deep inside a refrigerated metal box, pulling a mounted mouse-grown tumor incrementally closer to ...

A common pathway to cancer is when a protein responsible for fixing DNA mutations is itself non-functional. [Getty Images] Many people know that the causes of cancer are partly genetic. But how do ...